Research Teaching Specialist
Dr. Li Ni is a Research Teaching Specialist in the Reynolds Family Spine Laboratory, Department of Neurological Surgery at New Jersey Medical School. Dr. Ni graduated from Beijing Medical University, Beijing, China; and has 5 years of teaching experience in human anatomy and neuroanatomy prior to coming to USA in the late eighties. Trained as a neuroanatomist, Dr. Ni has extensive experience on in vivo and in vitro research techniques, such as immunohistochemistry, molecular biology, small animal surgery, cell culture, etc. Dr. Ni was involved in investigating various aspects of embryonic brain development for the last 15 years. In the laboratory he performs research on the immune response to acute spinal cord injury in mice.
Impaired sensitivity to pain stimuli in plasma membrane calcium ATPase 2 (PMCA2) heterozygous mice: a possible modality- and sex-specific role for PMCA2 in nociception.
Related Faculty & Staff: Veronika Khariv, M.S., Li Ni, M.D., Robert F. Heary, M.D., Stella Elkabes, Ph.D.
Toll like receptor 9 antagonism modulates spinal cord neuronal function and survival: Direct versus astrocyte-mediated mechanisms.
Related faculty and staff: Cigdem Acioglu, Ph.D., Ersilia Mirabelli, M.S., Li Ni, M.D., Robert F. Heary, M.D., Stella Elkabes, Ph.D.
More publications for Li Ni, M.D.
- Fifth Annual ‘Current Advances in Spinal Cord Injury Research’ Symposium, May 13th, 2015
- The second annual ‘Current Advances in Spinal Cord Injury Research’ Symposium
- The therapeutic potential of TLR ligands in spinal cord injury
- Proteomic strategies to define new therapeutic targets in animal models of spinal cord injury and disease
- Etiology of multiple sclerosis using an animal model of the disease
- Role of TLR9 in central nervous system development
- Neuron-glia interactions in the spinal cord
- Neuroprotection in spinal cord injury and disease
- Modulation of the inflammatory response in spinal cord injury and disease
- Early surgical decompression for the treatment of acute traumatic spinal cord injury
- The role of plasma membrane calcium ATPase 2 (PMCA2) in the processing of nociceptive and neuropathic pain signals in the spinal cord