Co-Director, Reynolds Family Spine Laboratory
Dr. Elkabes graduated from Bogazici (Bosphorous) University in Istanbul, Turkey with a Bachelor of Science degree in Chemistry. She received her M.S. and Ph.D. degrees in Neuroendocrinology from the Weizmann Institute of Science in Rehovot, Israel where she was awarded the Feinberg Graduate School Award for Excellency. Dr. Elkabes pursued her post-doctoral training in neurobiology at the National Institute of Health and Cornell University Medical College. She was a visiting scientist in the Department of Pharmacology, University of Milan, Italy. She held a faculty appointment in the Department of Neuroscience and Cell Biology, Robert W. Johnson Medical School, University of Medicine and Dentistry of New Jersey, before joining the faculty at New Jersey Medical School, Rutgers. She became the co-director of The Reynolds Family Spine Laboratory in the Department of Neurological Surgery at NJMS-Rutgers in 2010.
Dr. Elkabes’ research focuses on neuroprotection, neural regeneration, neuro-immune interactions and the cross-talk between neurons and glia in animal models of spinal cord injury and multiple sclerosis. Her research has been funded by various agencies including the National Institute of Health and New Jersey Commission on Spinal Cord Research. Dr. Elkabes has been a reviewer in NIH study sections since 2008. She has served as a reviewer in national and international funding agencies including The Israel Science Foundation; The Marsden Fund Council, New Zealand; The Medical Research Council, United Kingdom and Veteran Affairs, Department of Defense. Dr. Elkabes has served as ad-hoc reviewer in numerous scientific journals.
Dr. Elkabes is a full member of the Graduate School of Biomedical Sciences (GSBS) at NJMS-Rutgers. She received the Golden Axon Award for excellence in neuroscience teaching at NJMS. She has served as an acting co-director, executive committee member, admission committee member and the first year advisor of the Integrative Neuroscience Graduate Program, NJMS-Rutgers University joined graduate program in Newark, NJ. She is currently a member of the Cell Biology, Neuroscience and Physiology Track committee in the GSBS at NJMS-Rutgers.
Effects of early surgical decompression on functional and histological outcomes after severe experimental thoracic spinal cord injury.
Journal of Neurosurgery. Spine, January 2017.
Toll like receptor 9 antagonism modulates spinal cord neuronal function and survival: Direct versus astrocyte-mediated mechanisms.
Brain, Behavior, and Immunity, August 2016.
Delayed activation of human microglial cells by high dose ionizing radiation.
Brain Research, September 2016.
Dysfunction in amygdala-prefrontal plasticity and extinction-resistant avoidance: A model for anxiety disorder vulnerability.
Experimental Neurology, January 2016.
Chronic tissue response to untethered microelectrode implants in the rat brain and spinal cord.
Journal of Neural Engineering, February 2015.
Toll-like receptor 4 enhancement of non-NMDA synaptic currents increases dentate excitability after brain injury.
Neurobiology of Disease, February 2015.
Sex steroids and neuroprotection in spinal cord injury: A review of preclinical investigations.
Experimental Neurology, September 2014.
A toll-like receptor 9 antagonist improves bladder function and white matter sparing in spinal cord injury.
Journal of Neurotrama, November 2014.
Toll-like receptors in central nervous system injury and disease: a focus of the spinal cord.
Brain, Behavior, and Immunity, November 2014.
Maternal immune stimulation during pregnancy shapes the immunological phenotype of offspring.
Brain, Behavior, and Immunity, October 2013.
Toll-like receptor 9 deficiency impacts sensory and motor behaviors.
Brain, Behavior, and Immunity, August 2013
A toll-like receptor 9 antagonist reduces pain hypersensitivity and the inflammatory response in spinal cord injury.
Neurobiology of Disease, June 2013.
Pro-Inflammatory Phenotype Induced by Maternal Immune Stimulation During Pregnancy
Recent Advances in Autism Spectrum Disorders, InTech, March 2013.
Proteomic identification of immunoproteasome accumulation in formalin-fixed rodent spinal cords with experimental autoimmune encephalomyelitis.
Journal of Proteome Research, March 2012.
Reduced expression of plasma membrane calcium ATPase 2 and collapsin response mediator protein 1 promotes death of spinal cord neurons.
Cell Death and Differentiation, September 2010.
Role of plasma membrane calcium ATPase 2 in spinal cord pathology.
World Journal of Biological Chemistry, May 2010.
More publications for Stella Elkabes.
- Fourth Annual ‘Current Advances in Spinal Cord Injury Research’ Symposium, May 7th, 2014
- Sixth Annual ‘Current Advances in Spinal Cord Injury Research’ Symposium, May 11th, 2016
- Fifth Annual ‘Current Advances in Spinal Cord Injury Research’ Symposium, May 13th, 2015
- Third Annual ‘Current Advances in Spinal Cord Injury Research’ Symposium, May 8th, 2013
- Stella Elkabes, PhD receives a research grant from New Jersey Commission on Spinal Cord Injury Research
- The second annual ‘Current Advances in Spinal Cord Injury Research’ Symposium
- Inaugural ‘Current Advances in Spinal Cord Injury Research’ Symposium, May 11th, 2011
- The therapeutic potential of TLR ligands in spinal cord injury
- Proteomic strategies to define new therapeutic targets in animal models of spinal cord injury and disease
- Etiology of multiple sclerosis using an animal model of the disease
- Role of TLR9 in central nervous system development
- Neuron-glia interactions in the spinal cord
- Neuroprotection in spinal cord injury and disease
- Modulation of the inflammatory response in spinal cord injury and disease
- Early surgical decompression for the treatment of acute traumatic spinal cord injury
- The role of plasma membrane calcium ATPase 2 (PMCA2) in the processing of nociceptive and neuropathic pain signals in the spinal cord